Keywords: cerebral arteriosclerosis

        Cerebral arteriosclerosis chronic degenerative and proliferative changes of the cerebral arteries. Cerebral arteriosclerosis, cerebral arteriosclerosis is not exactly the same thing, there is a difference in the concept. Clinical manifestations and pathological examination, not only the degree of difference, and the nature of the difference. Only when severe cerebral arteriosclerosis, causing widespread cerebrovascular reduced blood flow and to widely affect brain function, and the unique clinical symptoms, known as cerebral arteriosclerosis. Cerebral arteriosclerosis necessarily have cerebral arteriosclerosis.

    Pathology on cerebral arteriosclerosis divided into three types: First, atherosclerosis, diffuse arteriosclerosis, hyalinization and fibrosis. Atherosclerosis occur mostly in the large arteries and middle artery intravascular lipid deposition. Diffuse atherosclerosis is more common in the arterioles. Hyalinization and fibrosis occurs mainly in the arterioles and capillaries. These three different types of pathology caused by the degeneration of the arterial wall, summed up collectively referred to as cerebral arteriosclerosis.

    Cerebral arteriosclerosis, the formation of a variety of factors, common factors are diabetes, lipid metabolism disorders, hypertension, smoking, obesity, endocrine disorders, mental factors.

    The formation of cerebral arteriosclerosis is a progressive slow process, there will be some early signals in the development process. Common early signal:

    ⑴ patients with similar symptoms of neurasthenia, such as dizziness, headache, stretching and head compression flu may also have tinnitus, vertigo, insomnia, memory loss (especially in recent memory impairment obviously), difficulty concentrating, easy fatigue, drowsiness, mood volatile, slow thinking, and is often accompanied by autonomic nervous system dysfunction, such as sweating, cold hands and feet, heart palpitations and other. Which, recent memory impairment is most characteristic of the early signals.

    ⑵ fundus examination revealed retinal artery significantly thinner reflective enhanced even with silver filamentous and dynamic, vein cross oppression.

    (3) Rheoencephalogram found significantly increased vascular tone and elasticity of blood vessels was significantly diminished.

    ⑷ isotopic determination of cerebral blood flow in the cerebral blood flow slows down and multifocal hypoperfusion area.

    ⑸ transcranial Doppler display cerebral artery stenosis performance.