Keywords: cerebral infarction clinical manifestations

        Cerebral infarction seen in more than middle-aged people, the majority of high blood pressure, diabetes, heart disease or a history of high cholesterol, and some of this has happened before TIAs or stroke. Usually acute onset and development reached a peak within a few hours. The proportion of patients in the early morning awoke to find an exception. Sick side headache, very little drama headaches, vomiting onset. Mainly the following four categories.

    (A) large artery atherosclerosis cerebral infarction in atherosclerotic arterial wall based on the development of blood clots or emboli, causing cerebral cortex diseased artery or hemispheres, cerebellum, brainstem local function loss. Specific symptoms, see above. Have the same arterial system the TIAs history. Continued deterioration of the small number of patients in the 24 hours after the onset or ladder-like heavier, may be due to the progressive extension of the thrombus or shedding new emboli.

    (B) heart tied infarction were more common in young adults, all of sudden onset, can be stepped increase. Often other cerebral artery TIAs, history of stroke or systemic embolism history. There is a cause of cardiogenic embolism.

    (C) lacunar infarction or small artery occlusive infarction development is relatively slow, and some can be gradually increased and reached its peak during up to 36 hours. Infarct volume, according to the occurrence site specific focal symptoms.

    (1) pure motor stroke: on the side, arm, leg, foot, toe paralysis, the PLIC or bridge, in the ventral small infarcts.

    (2) pure sensory stroke: the contralateral body’s feeling abnormalities seen in the ventrolateral thalamus lacunar.

    (3) ataxia laterality paresis: contralateral arm, hand Masonic disorder with leg paresis, seen in ventral pontine infarction.

    (4) dysarthria – clumsy hand: slurred speech and contralateral hand movement disorder for knee ventral pons or internal capsule infarction.

    (5) with the expression of the laterality of the aphasia knee and forelimb paresis: internal capsule infarction involving the white matter adjacent corona radiata.

    (D) due to other causes of cerebral infarction non-arteriosclerotic vascular disease, blood disease, blood clotting abnormalities and other rare causes of ischemic stroke.